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found that MEL can regulate neuroinflammation through activation of the Nrf2 pathway (136), another experimental and clinical study confirmed the protective effect of MEL on glial cells and its beneficial effect on DPN, but the specific therapeutic regimen of administration needs to be further clarified (137)

It is likely that the gloB mutant was more tolerant to MG due to the functional activation of potassium efflux pumps and protective cytoplasmic acidification

53, # 4, p

Finally CDAA-fed HGKO mice exhibited a significant reduction in systemic inflammation, as assessed by reduced circulating levels of IFN, IL-6 and TNF (Fig

doi: 10.1016/s0891-5849(02)01327-8

Side-effect peak at each dose increase, titrating down within 5-10 days at each stable step