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What is known so far is that cytokines-mediated neuroinflammation leads to dysregulations in the HPA axis, where the central regulator is cortisol releasing factor (CRF)

Sexual Dimorphism in Glucocorticoid Stress Response

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However, it had no effect on DEM-induced glutathione synthesis

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However, the same clinical trial revealed that only 6.2% of patients maintained any benefit at 6 months post-treatment compared to 0% in the placebo group