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In addition, the mitochondrial Ca 2+ uniporter (MCU) and mitochondrial permeability transition pore (mPTP) have been proposed as pathways for iron entry into mitochondria [87, 88], and blocking MCU by Ru360 reduced mitochondrial iron uptake and showed higher efficacy than blocking the mPTP with cyclosporin A as it leads to more profound decreases mitochondrial ROS levels and increased mitochondrial function, indicating that MCU may be a crucial portal for iron uptake by mitochondria [89]

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Neuron 82, 10171031

Abstract In autoimmune rheumatic diseases, immune hyperactivity and chronic inflammation associate with immune dysregulation and the breakdown of immune self-tolerance

Inhibition of proteinase 3 by [alpha]1-antitrypsin in vitro predicts very fast inhibition in vivo

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