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Restoration of myocellular copper-trafficking proteins and mitochondrial copper enzymes repairs cardiac function in rats with diabetes-evoked heart failure

After this period, the vial should be discarded

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Thus, these altered metabolomic changes and perturbed balance in the motor cortex of prpTDP-43 A315T mice suggest a deficit in energy homeostasis and increased oxidative stress, events that are associated with problems with mitochondrial integrity and function

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