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HMGB1, in particular, can be actively released under certain stimuli, as well as passively released during inflammatory cell death processes (119)

10.1128/IAI.72.10.5750-5758.2004

The upregulation of these immunes-related pathways delineated above was attributed to the Ba/O 2 -supplied radiosensitization and multistage immune activation by sequential aCTLA-4/IL-12 release