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doi: 10.1007/s12272-013-0110-x

David LA, Weil A, Ryan ET, Calderwood SB, Harris JB, Chowdhury F, et al

Since in hypertensive patients the low levels of GSH are often accompanied by a decrease of companion enzymes (catalase, GSH-peroxidase and GST) and increased lipid peroxides, the prevailing hypothesis is that the GSH decrease is due to a burst of ROS production, secondary to an inflammatory process [76]

In contrast to autophagy, ferroptosis does not have the formation of classical closed bilayer membrane structures (autophagic vacuoles)

doi: 10.3390/molecules28124713

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