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Nonetheless, the interpretation of these studies is complicated by the fact that these models are constitutive genetic deletions, and due to the lack of FA mobilization from adipocytes, these mice are completely reliant on glucose as a fuel source

This problem is closely associated with a genetic component

Frameshift mutations of autophagy-related genes ATG2B, ATG5, ATG9B and ATG12 in gastric and colorectal cancers with microsatellite instability

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