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In addition, our observation that higher IGF-1 concentration is associated with longer survival is consistent with experimental research, showing that IGF-1 acts as a mitochondrial protector in the ALS cell and mouse model 10

in order to limit time to freezing (and potential changes in metabolic phenotype due to death) no additional processing of tissue occurred and organs were not perfused prior to collection

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Dopamine-induced protection against indomethacin-evoked intestinal lesions in ratsrole of anti-intestinal motility mediated by D2 receptors

Several antioxidant therapies have been found to delay cataract progression