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In contrast, degradation of VDAC2/3 and carbonylation of VDAC2 by RSL3 have been reported [42, 43], suggesting the potential role of VDAC in RSL3-mediated ferroptosis depending on cell types or context

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Consequently, signaling pathways such as mitogen-activated protein kinase (MAPK) signaling and tyrosine kinase activity were altered, leading to modulation of kinases and promoting aggressive breast cancer behavior

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