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GH and IGF-1 both play roles in tissue repair, collagen synthesis, and cellular regeneration

The crosstalk between distinct RCDs is well documented and can be interpreted through the lens of the cell death-governing intracellular system with multiple redundant backup pathways [20, 142]

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In terms of mechanism, DNA damage repair is the most critical reason for the radiotherapy resistance of GH and IGF1 in cancer cells and damage repair effect on adjacent tissues

mVEGFR1 trafficking and stability depends on Rab11a, a small GTPase associated with the slow recycling of proteins between endosomes and the cell surface, similar to D6 (refs 63, 64)

Rapid control of severe hypertension with minoxidil