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Despite the fact that in most cases UGTs are responsible for glucuronidation of their substrates, members of the UGT1A subfamily have been found to catalyze glucuronidation of several aminecontaining substrates (chlorpromazine, amitryptyline) (Tukey & Strassburg, 2000)

Previous research has shown that glycolysis, as the main energy source for tumors, has a significant impact on the initiation of AML, as well as the survival, proliferation, invasiveness, and drug resistance of AML cells [39, 40]

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NHS (2016)

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