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Excessive reactive oxygen species (ROS) disrupt cellular redox homeostasis and directly induce oxidative modifications (e.g., carbonylation, disulfide bond disruption) in tight junction proteins, compromising gastrointestinal barrier integrity and increasing intestinal permeability

While the benefits can be rewarding, patient safety must always come first

Dantas TL, Alonso Buriti FC, Florentino ER

Anti-cerebral ischemia reperfusion injury of polysaccharides: A review of the mechanisms

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The inflammatory cascade reaction after ischemia is produced by multiple cytokines and not by a single cytokine