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Its different effects on lipoprotein metabolism with a possible role in the modulation of inflammation makes the net impact of apoC1 on cardiometabolic risk difficult to figure out and apoC1 might be considered as pro-atherogenic or anti-atherogenic depending on the overall metabolic context

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observed that TNBC-derived C-X-C motif chemokines 1/2/8 (CXCL1/2/8) stimulate CAFs and other lung-resident fibroblasts to secrete C-C motif chemokines 2/7 (CCL2/7), thereby activating cholesterol synthesis in TNBC cells to support metastatic tumor growth [30]

[12] LptE forms a complex with LptD, which is involved in the assembly of LPS in the outer leaflet of the outer membrane and is essential for envelope biogenesis

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