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The results of these analyses in old mice supplemented with GlyNAC were compared with those of the old-control group and with the corresponding data obtained from young mice

Although the molecular mechanisms underlying these differences are not clear, data from experimental models suggests that estrogen deficiency results in the activation of several regulatory mechanisms such as the renin-angiotensin-aldosterone axis, which lead to the development of cardiac hypertrophy and fibrosis (Benjamin et al., Aging-Related Heart Alterations Aging-associated cardiac remodeling is characterized by increased oxygen and energy demand due to elevated afterload and hypertrophy of cardiomyocytes (Lakatta and Levy, 2003b)

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doi: 10.1158/1078-0432.CCR-20-4521 59 MahrusSKisielWCraikCS

Organization of -adrenoceptor signaling compartments by sympathetic innervation of cardiac myocytes

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