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Another future direction of utilising GLP-1/GIP/glucagon receptor triagonists is for MASH therapy with the rationale that as GLP-1 receptor agonists halt fibrosis progression, the new therapies might be able to enable fibrosis regression [11]

P., Schneider M., Proneth B., et al

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Consistently, Bis-2-(5-phenylacetamido-1,3,4-thiadiazol-2-yl)ethyl sulfide (BETPS), a GLS1-specific inhibitor (Robinson et al., 2007), failed to block serum-dependent necrosis in MEFs (Figure S5C)

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Maintaining proper storage conditions is essential for ensuring the medication remains stable and suitable for use throughout its beyond-use date