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NF-B regulates many genes in the inflammatory response that can be promoted by proinflammatory stimuli, such as CS and TNF- ( in vivo experiment showed that GHK-Cu blocked the transcriptional activity of NF-B through suppression of NF-B by altering the phosphorylation of its inhibitor i-B

2012c;367(6):542-550

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A total of 71 patients were included in the study
