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By analysing cf-mtDNA levels in the tear fluid, mitochondrial damage and impairments can be detected early at the stage of reversible damage to health, which is crucial for tailoring preventive measures and individualised treatments [36, 66, 71, 72]

Some studies, however, suggested that ebselen is an inefficient catalyst due to formation of various unreactive intermediates that prevent the regeneration of the original compound (Sarma and Mugesh, 2005

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Indeed, while we anticipate some of these therapies may be broadly useful to any individual with a definite PMD, we recognize that in the growing era of precision medicine a personalized approach to tailor the precise therapeutic selection to each patients broader genomic or metabolic profile and specific molecular diagnosis (Table 2)