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10.5489/cuaj.8685 5 AshkarA.SosnikA.Davidovich-PinhasM

Studies on the treatment of chronic myelogenous leukemia (CML) have revealed that resistance to imatinib develops after long-term use and that inhibition of BCR-ABL with imatinib or MAPK/cholesterol esterification via avasimibe treatment alone led to insufficient effects

Cellular model of Alzheimers disease: A1-42 peptide induces amyloid deposition and a decrease in topo isomerase II

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GLP-1 signalling slows gastric emptying and blunts appetite centrally

Further molecular refinement revealed the enrichment of missense mutations and deletions in histone-lysine N-methyltransferase 2A ( KMT2A ) or MLL and histone deacetylase ( HDAC ) family genes within Cluster M2, underscoring the role of chromatin remodeling in GBM pathogenesis