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From DNA targeting to covalent modification of proteins, and metabolic regulation just to highlight a few

NTHi invasion of host epithelial cells may not only provide a means of evading host immune pressures, but also provide an alternative reservoir for nutrient acquisition when bacteria are exposed to nutrient-limiting conditions ( sapA mutants deficient in haem-iron uptake demonstrated a tendency towards a more persistent phenotype, favouring intracellular survival and a dampened cytokine response compared to the parent strain (Raffel et al., 2013)

Impaired TRPM3-Dependent Calcium Influx and Restoration Using Naltrexone in Natural Killer Cells of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome Patients. Journal of Translational Medicine, vol