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Neuropsychiatric effects have been reported in post-marketing surveillance of GLP-1 receptor agonists, though the incidence and causal relationship remain unclear

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The evidence ranges from preclinical animal studies to human clinical trials, and the mechanisms are diverse enough that different peptides address different aspects of nerve damage

[36] Shivering and diarrhea are mild serotonin syndrome symptoms, but rigid muscles, fever, and seizures are severe symptoms

however, our data show that BACH1 stabilization promotes angiogenesis, which may be attributed to cell type specificity or context-dependent downstream targets (e.g., PDGFR instead of HO-1) [17, 18]