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This metabolic dysregulation is defined by systemically reduced levels of metabolites such as amino acids and fatty acids, leading to reduced TCA cycle activity and oxidative phosphorylation, which may ultimately be a major contributor to neuropsychiatric symptoms associated with reduced energy, such as fatigue and other cognitive impairments [43, 62]

Since then, the idea of metabolic syndrome (MetS) has been established [21]

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Available genetic and biochemical studies suggest that adipose tissue plays a key role in the development of insulin resistance [51]