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[DOI] [PMC free article] [PubMed] [Google Scholar] Wang G, Hu C, Zhou J, et al

In wild-type mice, Cx32 abundance in hepatocytes decreased substantially as early as 1-h post-APAP treatment, reaching a 20% reduction compared to the steady-state or control group

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The liver also has a unique property that allows it to regenerate itself in cases of damage

Interestingly, blocking NETs does not affect steatosis and free fatty acid accumulation but inhibits HCC development (68)

While the evidence is limited, HSR activation downstream of mild heat shock appears to be less context-dependent than UPR activation