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explored the acute and subchronic oral toxicity of Ni NPs in rats [147]

56 Collectively, various intracellular and extracellular stimuli contribute to ROS formation in cancer cells (Fig

However, the mechanism of KD benefits is still not fully understood, despite some evidence indicating its multiple targets and ability to regulate histone deacetylase, glycolysis, neurotransmitter levels, the function of NLRP3 inflammasome, and oxidative stress

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In silico ADMET examination disclosed that all the ligand molecules have no toxic effect and acceptable absorption as well

Oxidative stress stimulates utilization of the methylglyoxal pathway
