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Coincidentally, among these genes, maoc-1 (MAO-C-like dehydratase domain), dhs-28 (dehydrogenases, short chain), dauer formation-22 ( daf-22 ), and peroxisome assembly factor-10 ( prx-10 ) have been reported to play important roles in the peroxisomal -oxidation pathway, and their absence may cause the excessive accumulation of TAG [49]

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