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Most mammalian models express functional UOX, which prevents the spontaneous development of persistent hyperuricemia and requires exogenous interventions
doi: 10.1038/nrneph.2016.122 2 AhmedA
doi: 10.3324/haematol.2018.212977
Ethics declarations Conflict of interest The UMCG, which employs several of the authors has received research grants and/or fees from AstraZeneca, Abbott, Boehringer Ingelheim, Cardior Pharmaceuticals Gmbh, Ionis Pharmaceuticals, Inc., Novo Nordisk, Vifor, and Roche
Superoxide radical production in response to environmental hypoxia in cultured shrimp
An LKB1mitochondria axis controls TH17 effector function
