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Figure 12 Using stLearn, we visualized the spatial coexpression of FN1 and ITGB1 across all spots, significant spots, and annotated regions, revealing spatially confined ligandreceptor activity ( Figure 12E )

(Compositae) was first reported to protect liver against APAP-induced hepatotoxicity (Gilani and Janbaz, 1993)

"ASCO guideline: chemotherapy-induced peripheral neuropathy." J Clin Oncol 2020;38(28):3325-48

Estrogen Metabolism Support Optimizing hepatic Phase I and Phase II estrogen detoxification reduces the burden of potentially carcinogenic estrogen metabolites (particularly 4-OH and 16-OH estrone) and may reduce estrogen-driven fibroid growth: DIM (diindolylmethane): Shifts estrogen metabolism toward the favourable 2-OH pathway Calcium-D-glucarate: Inhibits -glucuronidase, preventing deconjugation and reabsorption of estrogens in the gut (Note: off-the-shelf detox kits from health food stores are not equivalent to therapeutic doses of these specific compounds) N-acetylcysteine (NAC): Supports glutathione synthesis and Phase II sulphation 3

The presence of severe hepatic steatosis has been shown to have implications on metabolic complications, including metabolic syndrome, and thus, these patients should be monitored for the development of complications, especially since metabolic risk factors, including weight and dysglycemia, can increase over time