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Glia 69 , 17491766 (2021)

While this research is extensive and consistently positive, the gap between animal and human studies represents both a limitation of current knowledge and an opportunity for future research

PUFA-PLs are susceptible to lipid peroxidation induced by arachidonic acid (A)LOXs, which eventually results in the loss of the lipid bilayer and impairs cell membrane function, thus increasing ferroptosis (49)

1b).Therefore, we have used N2a cells as a model system of immature neurons

Antioxid Redox Signal 15: 1583-1606

Targeting pathways including NCOA4 may be a promising approach for the treatment of glioma