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[DOI] [Google Scholar] Aziz, M

Others have shown that cellular oxidative stress contributes to dysregulation of both pro-oxidant (NOX enzymes) and anti-oxidant enzymes (catalase, superoxide dismutases, peroxiredoxins, and glutathione peroxidases) [47] and this could be seen in the neutrophil genomic changes in our study predominantly in redox-associated genes

After mitophagy induction, autophagolysosomes breakdown antioxidant substances and ferritin [123], allowing for the release of free Fe 2+ and the Fenton reaction to proceed with unreduced ROS [124]

Furthermore, LicoA inhibited cisplatin-stimulated lipid peroxidation (Figures 3FH)

PQQ is highlighted for its significant role in mitochondrial function, being 100 to 1,000 times more potent than CoQ10

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