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Description
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Biological systems are not additive
BCL-2, the proto-oncogene, has the ability to block apoptosis, leading to reduced cell death and an enhanced proliferation (Omar et al., 2019)
These fatty acids are catalyzed by acyl-coenzyme A synthetase long chain family member 4 (ACSL4) and converted to PUFA-coenzyme A (CoA) [31]
NETosis promotes chronic inflammation and cancer progression by inducing DNA damage, proliferation, and angiogenesis via MMP9 and VEGF
Potential long-term effects could include joint pain and increased risk of diabetes due to altered glucose metabolism
