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Maladaptive dendritic spine remodeling contributes to diabetic neuropathic pain

While they sound alike, their primary "workstations" in the body are different

Typically, -synuclein localizes to the mitochondria-associated membrane, and its presence may disrupt mitochondrial function by promoting the formation of the mitochondrial permeability transition pore (mPTP), leading to mitochondrial membrane potential (MMP) loss, subsequent mitochondrial degradation, and ultimately cell death (192)

Baloglu, M.E

Furthermore, they downregulate the expression of nuclear factor-kappa B (NF-B), a master genetic switch that controls the production of severe inflammatory cytokines like Interleukin-6 (IL-6) and Tumor Necrosis Factor-alpha (TNF-)

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