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The role of endothelial dysfunction and oxidative stress in cerebrovascular diseases

The first evidence showing APP overexpression in DS was demonstrated within enlarged endosomes and lysosomes, where APP accumulation could be initiated at the fetal stage, as early as 28 wk of gestation (465)

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Chlorophyll binds to toxins and deactivates them by preventing them from binding to DNA and cellular receptors, decreasing the cellular damage caused by drinking

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Cellular Protection and Angiogenesis studies of copper-mediated antioxidant enzyme activation and vascular regeneration
