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Diverse stress signals produced during tumor initiation, such as those emitted by telomere attrition, DNA damage, nutrient deprivation, hypoxia, oxidative stress, and hyperproliferation, etc , can activate p53 and initiate the p53-dependent tumor suppression responses such as DNA repair, cell-cycle inhibition, senescence, autophagy, apoptosis [59]

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[DOI] [PMC free article] [PubMed] [Google Scholar] 22.Hou CC, Lin H, Chang CP, Huang WT, Lin MT

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