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used dihydroartemisinin to intervene in a fibrotic cell model established using a human embryonic lung fibroblast cell line and found that dihydroartemisinin inhibited cell viability, decreased Fe 2+ levels, and inhibited the expression of ferritin heavy chain 1 (FTH1) and nuclear receptor coactivator 4 (NCOA4) genes and proteins

Compared to Ad cells, significantly increased 13 C 3 -lactate-derived metabolites in AR cells included pyruvate, acetyl-CoA, TCA cycle intermediates (fold change from high to low: oxaloacetate, succinate, -ketoglutarate, malate, fumarate, isocitrate, and citrate), and TCA cycle-derived metabolites (glutamate, aspartate, and ribose-5-phosphate), indicating enhanced lactate uptake and oxidation as well as increased TCA metabolism (Fig

Oral NR supplementation in aged participants elevates the muscle NAD + metabolome, ameliorates metabolic dysfunction, depresses levels of circulating inflammatory cytokines and increases the anti-inflammatory molecule adiponectin in aged human

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[] ASBT INHIBITORS Like bile acid resins, apical sodium-dependent bile acid transporter (ASBT, SLC10A2) inhibitors attenuate bile acid reabsorption and hepatobiliary bile acid toxicity