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[DOI] [PubMed] [Google Scholar] 47.Pellissier L.P., Pujol C.N., Becker J.A.J., Le Merrer J

One of the first requirements is a clear scientific rationale explaining why the compound's mechanism of action or biological target may potentially provide a therapeutic benefit

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The inhibition of HDAC1 expression increases the expression of DUSP1, significantly reduces fibrosis markers, and hinders the phosphorylation of Smad3, thus alleviating UUO-induced renal tubules injury and renal fibrosis (Wang S

Across the usual run of cell-counting, MTT, and BrdU assays, fibroblast proliferation tends to rise by something like 15% to 40% over untreated cells