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[7] last access: 11.01.2023 [8] last access: 11.01.2023 [9] last access: 11.01.2023 [10] Buck, MJ, Lieb, JD

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The arteries of Sm22 / mice develop enhanced inflammatory response and oxidative stress, which contributes to neointimal hyperplasia through different signaling mechanisms, 41,42,43 suggesting that Sm22 / VSMCs have transited to a synthetic or pro-inflammatory state

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NEK2, AURKC, RPS6KA2, PAK2, and PLK1 are also involved in cell cycle regulation reviewed in (5862) and are predicted to have significantly increased kinase activity
