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[DOI] [PubMed] [Google Scholar] 146.Chen Y., Zhang J., Zhang T., Wu Y., Xi Y., Wu T., Li M., Li Y., Zhou S., Wu M., et al

Figure 2 2.1 Reproductive system inflammation in PCOS 2.1.1 Ovary SLCI contributes to defects in oocyte quality, leading to ovulatory infertility, and accelerating the decline in ovarian reserve ( in vitro fertilization and embryo transfer (IVF-ET) treatment, reflecting the local function and pathological state within the PCOS ovaries ( Pro-inflammatory TNF-, primarily secreted by macrophages, was significantly increased in the ovaries and FF of PCOS patients ( In vitro experiments demonstrated that TNF- can directly induce apoptosis of primary rat GCs after 24h of incubation in serum-free medium 24 ( in vivo treatment with TNF inhibits steroidogenesis of the thecal cells and GCs, leading to regression of the corpus luteal ( Another pro-inflammatory cytokine IL-6, which is produced by mononuclear (MNC) cells and adipose tissue cells

All three cell types can interact with the others and fuel a vicious activation circle (164)

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J Cell Mol Med 20 , 769781.CrossRefGoogle ScholarPubMed 15 , 107560.CrossRefGoogle ScholarPubMed In vivo NMR studies of the glutamate neurotransmitter flux and neuroenergetics: implications for brain function