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This process not only directly generates excessive ROS by initiating the non-enzymatic Fenton reaction but also enhances the activity of lipoxygenase (ALOX) or EGLN prolyl hydroxylases (also known as PHD) [1, 19, 20]

Malignant transformation of cancer exhibits high intrinsic generation of ROS level which controls the tumorigenic phenotype and propels the tumor progression

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Brust, M., Walker, M., Bethell, D., Schiffrin, D

Role of phenolic compounds in peptic ulcer: An overview