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One common oxidative DNA lesion is 8-hydroxydeoxyguanosine (8-OHdG), which results from the interaction of ROS with guanine bases [97]
Furthermore, the gliotoxin treatment also reduced the DNA-binding activity of NF-B, thus impeding the inflammation mediated by NF-B activation in the colon [30]
Peter, K., Rehli, M., Singer, K., Renner-Sattler, K
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Journal of Molecular Medicine
Narsale A, Moya R, Davies JD
