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Since mitochondria are essential to our very existence, it is unsurprising that the effects of mitochondrial diseases can be severe, and sometimes lethal, spanning a wide-ranging spectrum of physiological and biochemical symptoms [1,3,4] (see Table 1 for examples)

3.11 Neuroprotective efficacy of GO@GSH-FA on neuronal injury post-ischemia-reperfusion To elucidate the neuroprotective potential of GO@GSH-FA, we performed both histological and molecular analyses on cerebral ischemia-reperfusion injury lesions

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However, following treatment with NAC, insulin content and insulin mRNA expression were found to be preserved, and the binding of the nuclear factor pancreatic-duodenal homeobox-1 (PDX-1) to insulin was also restored

BMC Complement Med Ther 24(1):193 Harjumki R, Pridgeon CS, Ingelman-Sundberg M (2021) CYP2E1 in alcoholic and non-alcoholic liver injury