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A prominent mechanism by which curcumin exerts antitumor activity is through inhibition of the phosphoinositide 3-kinase/protein kinase B/mammalian target of rapamycin axis, a signaling hub critically involved in prostate tumorigenesis, particularly in phosphatase and tensin homolog-deficient contexts (De Velasco et al., 2020)

Notably, MFN18 elevated lipid accumulation and negated the beneficial effect of -OHB on lipid deposition (Fig

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Presenting the antigen in this way regulates gluten-specific T cell activity to rewire the immune response to gluten