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Various pharmaceuticals, including erastin and RSL3, provoke ferroptosis by diminishing glutathione peroxidase 4 (GPX4) activity, facilitating the accumulation of lipid peroxides [88]

[DOI] [PMC free article] [PubMed] [Google Scholar] Brodzikowska A., Ciechanowska M., Kopka M., Stachura A., Wodarski P.K

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STING Aggravates Ferroptosis-Dependent Myocardial Ischemia-Reperfusion Injury by Targeting GPX4 for Autophagic Degradation

The metabolic function of the peroxisome relies on its interaction with various subcellular organelles, including mitochondria, which play a role in re-oxidizing the NADH produced during the peroxisomal beta-oxidation [108]