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Similarly, PPAR activation in Ldlr / mice fed with HFD, which increases cardiac PPAR levels (51, 52), reduces Ppargc1a expression and causes cardiac hypertrophy (53)

suggest that GHK-Cu may also tone down inflammatory signaling in macrophages activated by pro-inflammatory triggers and in lung cell injury models

The ability of MK-677 to stimulate IGF-1 production can also indirectly improve cognitive function based on IGF-1s role in cognitive function

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"Thymosin beta 4 and a synthetic peptide containing its actin-binding domain promote dermal wound repair." J Invest Dermatol

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