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Even though the pro-inflammatory M1-like phenotype is regarded as anti-tumor macrophage and the anti-inflammatory M2-like state as tumor-promoting macrophage (15, 16), this is an oversimplified distinction as these cells are dynamic and heterogeneous within and across tumors

Chronic ethanol exposure resulted in pronounced structural alterations in both liver and pancreatic tissues, including hepatocyte degeneration, necrosis, and disruption of normal pancreatic architecture, reflects the extensive cellular and inflammatory stress induced by ethanol (Figures 9, 10)

Long-term HFD consumption reduced hepatic expression of glutaredoxins The de-glutathionylation of protein-mixed disulfides is primarily catalyzed by glutaredoxins (Grxs) 31